The first event in the inflammatory response consists in the recruitment of circulating monocytes and other myeloid cells to specific locations, such as bacterial entry sites, ischemic tissues, intratumoral areas, or atherosclerosis plaques. These pathological sites are normally associated with inadequate or abnormal vascularization, which subsequently compromises appropriate oxygen supply and metabolite exchange. It is now clear that metabolites, metabolic competition and nutrient restriction contribute to the immune response, determining disease outcome. This special issue of Immunometabolism aims to provide a series of articles covering all the aspects to point out how metabolic reprogramming and metabolic cross-talk control innate immunity and inflammatory cell phenotypes during acute and chronic inflammation such as cancer, ischemia, atherosclerosis and infections. We are welcoming submissions of both original research articles and review articles related to this topic.
Professor Massimiliano Mazzone
Myeloid-derived suppressor cells
Acidity and pH
Submission Deadline: 15 June 2019
Manuscripts should be submitted online through Hapres Online Submission System. Please visit Guide for Authors before submitting a manuscript. Authors are encouraged to submit a paper as soon as it is ready and don’t need to wait until the deadline. Submissions will be sent to peer-review in order of arrival. Accepted papers will be published continuously in Immunometabolism and then gathered together on the special issue webpage. We welcome Research articles, Review papers and Short Communications. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.
VIB, KU Leuven